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VitaDAO
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VitaDAO
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VitaDAO is a community owned collective funding early stage longevity research, governed by $VITA-holders
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VitaDAO
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New Vita-FAST proposal: VFDP-14 🔥 Team discovered commercially available compounds showing exceptional autophagy activity - the strongest they've tested. Now scoping a potential Phase 2 trial in UAE. $30K budget requested for IP filling, regulatory, and manufacturing consultants to assess feasibility from the VITA-FAST community
New Vita-FAST proposal: VFDP-14 🔥
Team discovered commercially available compounds showing exceptional autophagy activity - the strongest they've tested.
Now scoping a potential Phase 2 trial in UAE.
$30K budget requested for IP filling, regulatory, and manufacturing consultants to assess feasibility from the VITA-FAST community
VITA
Alpha
-2.46%
VitaDAO
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VitaLabs Projects Update
VitaLabs Projects Update
VitaDAO
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VitaDAO community call happening today at 5pm CET Join us for project updates from VitaLabs-funded researchers. They'll present their latest progress and answer your questions on the projects they're working on RSVP https://lu.ma/n06v3o81 👇
VitaDAO community call happening today at 5pm CET
Join us for project updates from VitaLabs-funded researchers.
They'll present their latest progress and answer your questions on the projects they're working on
RSVP https://lu.ma/n06v3o81 👇
VITA
Alpha
-2.46%
VitaDAO
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10 years of ethereum means 10 years of 'what if we could...' becoming 'we actually did' vitadao is just one of thousands of those stories happy birthday eth 🎂
10 years of ethereum means 10 years of 'what if we could...' becoming 'we actually did'
vitadao is just one of thousands of those stories
happy birthday eth 🎂
VITA
Alpha
-2.46%
VitaDAO
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VitaDAO community call Aug 1st, 5pm CET Join us for project updates from VitaLabs-funded researchers. They'll present their latest progress and answer your questions on the longevity science they're working on RSVP👇 https://lu.ma/n06v3o81
VitaDAO community call Aug 1st, 5pm CET
Join us for project updates from VitaLabs-funded researchers.
They'll present their latest progress and answer your questions on the longevity science they're working on
RSVP👇
https://lu.ma/n06v3o81
VITA
Alpha
-2.46%
VitaDAO
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accelerating to an ageless future
accelerating to an ageless future
VitaDAO
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some rules were meant to be broken aging is one of them
some rules were meant to be broken
aging is one of them
VITA
Alpha
-2.46%
VitaDAO
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science is catching up to science fiction soon
science is catching up to science fiction soon
VitaDAO
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Jellyfish reverse their aging when stressed lobsters produce telomerase forever trees live for thousands of years death isn't universal in nature it's just common in mammals let's make death optional
Jellyfish reverse their aging when stressed lobsters produce telomerase forever
trees live for thousands of years
death isn't universal in nature it's just common in mammals
let's make death optional
VitaDAO
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Your great-great-grandparents would think smartphones are magic. Your great-great-grandkids will think death was barbaric.
Your great-great-grandparents would think smartphones are magic.
Your great-great-grandkids will think death was barbaric.
VitaDAO
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from the mountain: cities evolving past death, one edit at a time longevity/acc means building futures where death is optional 🧬
from the mountain: cities evolving past death, one edit at a time
longevity/acc means building futures where death is optional 🧬
VitaDAO
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What if breakthrough connections in aging research are hiding in plain sight? Map it as a knowledge graph: 🌐 > NAD+ depletion triggers mitochondrial dysfunction. > Dysfunctional mitochondria drive cellular senescence. > Senescent cells secrete SASP factors causing inflammation > Inflammation depletes NAD+ further. Each node branches into hundreds more. Making death optional isn't in one breakthrough —it's in seeing how everything connects.
What if breakthrough connections in aging research are hiding in plain sight?
Map it as a knowledge graph: 🌐
> NAD+ depletion triggers mitochondrial dysfunction.
> Dysfunctional mitochondria drive cellular senescence. > Senescent cells secrete SASP factors causing inflammation
> Inflammation depletes NAD+ further.
Each node branches into hundreds more.
Making death optional isn't in one breakthrough —it's in seeing how everything connects.
VitaDAO
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The Thinker (2025): debugging it 🧬 Silicon synapses firing at light speed. Marble transformed to motherboard. Not thinking ABOUT existence—compiling the next version. This is your brain on longevity/acc 🔄
The Thinker (2025): debugging it 🧬
Silicon synapses firing at light speed. Marble transformed to motherboard.
Not thinking ABOUT existence—compiling the next version.
This is your brain on longevity/acc 🔄
VitaDAO
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Every disease was incurable until it wasn't. Every impossible breakthrough was science fiction until it wasn't. Aging is next.
Every disease was incurable until it wasn't.
Every impossible breakthrough was science fiction until it wasn't.
Aging is next.
VitaDAO
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Gero, backed by the VitaDAO community, just landed a $1B+ deal with Chugai (Roche)! Their AI finds the root causes of age-related diseases → Chugai develops antibody drugs from these discoveries. Big leap forward for longevity research 🧬 https://biospace.com/business/roches-chugai-breaks-into-aging-via-potential-1b-deal-with-ai-outfit-gero
Gero, backed by the VitaDAO community, just landed a $1B+ deal with Chugai (Roche)!
Their AI finds the root causes of age-related diseases → Chugai develops antibody drugs from these discoveries.
Big leap forward for longevity research 🧬
https://biospace.com/business/roches-chugai-breaks-into-aging-via-potential-1b-deal-with-ai-outfit-gero
VitaDAO
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https://t.co/HAMazJbe5H, backed by the VitaDAO community, just landed a $1B+ deal with Chugai (Roche)! Their AI finds the root causes of age-related diseases → Chugai develops antibody drugs from these discoveries. Big leap forward for longevity research 🧬
https://t.co/HAMazJbe5H, backed by the VitaDAO community, just landed a $1B+ deal with Chugai (Roche)!
Their AI finds the root causes of age-related diseases → Chugai develops antibody drugs from these discoveries.
Big leap forward for longevity research 🧬
VitaDAO
--
Senescent cells: The zombies in your body 🧟 They stop dividing but refuse to die, secreting inflammatory factors that age surrounding tissue. In young bodies, the immune system clears them. In older bodies, they accumulate. Time to bring in the cellular cleanup crew.
Senescent cells: The zombies in your body 🧟
They stop dividing but refuse to die, secreting inflammatory factors that age surrounding tissue.
In young bodies, the immune system clears them.
In older bodies, they accumulate.
Time to bring in the cellular cleanup crew.
VitaDAO
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Your grandchildren might never experience aging the way you do. Right now, labs worldwide are cracking the code on cellular rejuvenation, AI-driven drug discovery, and biological age reversal. We're funding the science that makes death optional
Your grandchildren might never experience aging the way you do.
Right now, labs worldwide are cracking the code on cellular rejuvenation, AI-driven drug discovery, and biological age reversal.
We're funding the science that makes death optional
VitaDAO
--
desci summer ▓▓▓▓▓▓▓▓▓▓░░░░ 70%
desci summer ▓▓▓▓▓▓▓▓▓▓░░░░ 70%
VitaDAO
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.@vitarnabio/@artanbio teams up with @Syenex for precision in vivo delivery of their lead drug ARTAN-102. 🎯Goal: Get their aging therapeutics exactly where they need to go - faster path to human trials ahead. 🫡 https://www.prnewswire.com/news-releases/artan-bio-partners-with-syenex-vivocell-platform-for-precision-in-vivo-delivery-of-next-generation-codon-suppressors-for-aging-302494079.html?tc=eml_cleartime
.@vitarnabio/@artanbio teams up with @Syenex for precision in vivo delivery of their lead drug ARTAN-102.
🎯Goal: Get their aging therapeutics exactly where they need to go - faster path to human trials ahead. 🫡
https://www.prnewswire.com/news-releases/artan-bio-partners-with-syenex-vivocell-platform-for-precision-in-vivo-delivery-of-next-generation-codon-suppressors-for-aging-302494079.html?tc=eml_cleartime
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